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topical anesthetic agents,
local anesthetics drugs
BP Standard Lidociane HCL Powder,99.86% Lidocaine HCL
Product Name: Lidocaine hydrochloride
Synonyms: 2-DIETHYLAMINO-N-(2'6'-DIMETHYLPHENYL)ACETAMIDE HCL;2-DIETHYLAMINO-N-[2,6-DIMETHYLPHENYL]ACETAMIDE HCL;ALPHA-(DIETHYLAMINO)-2',6'-ACETOXYLIDIDE HYDROCHLORIDE;A-(DIETHYLAMINO)-2 6-DIMETHYLACETANILIDE HYDROCHLORIDE;LIGNOCAINE HYDROCHLORIDE;LIDOCAINE HYDROCHLORIDE;LIDOCAINE HCL;2-(diethylamino)-n-(2,6-dimethylphenyl)-acetamidmonohydrochloride
Product Categories: API;Intermediates & Fine Chemicals;Pharmaceuticals;Amines;Aromatics;Research Chemical;Pharma materials
Mol File: 73-78-9.mol
Lidocaine hydrochloride Structure
Chemical Properties White to Off-White Solid
Usage Local anesthesic;Na+ channel blocker
Usage Anesthetic (local); antiarrhythmic (class IB). Long-acting, membrane stabilizing agent against ventricular arrhythmia. Originally developed as a local anesthetic.
Definition ChEBI: The anhydrous form of the hydrochloride salt of lidocaine.
Lidocaine hydrochloride for amide local anesthetics. After the absorption of blood or intravenous administration, there was biphasic excitatory and inhibitory effects on the central nervous system, and no precursor excited, blood concentration is low, analgesic and sleepiness, pain threshold increased; with the dose increase, enhancing effect or toxicity, have the anticonvulsant effect of inferior toxic blood concentration.
When the blood concentration of more than 5 mu g ml-1 convulsions may occur. The goods at low doses, can promote myocardial intracellular k+ outflow, reduce myocardial and self-discipline, has antiarrhythmic effect; in therapeutic doses, had no significant effect on the electrical activity of myocardial cells, the atrioventricular and myocardium contraction; blood drug concentration increased further, which can cause cardiac conduction velocity, atrioventricular block, inhibition of myocardial contractility and cardiac output decreased.
The goods after the injection, tissue distribution of fast and wide, can penetrate the blood-brain barrier and the placenta. The anesthesia of high strength, quick effect, dispersion force, from the local drug elimination takes about 2 hours, plus epinephrine can prolong the action time. Most of the first by the enzyme degradation of liver microsomes for local anesthesia is still used deethylase intermediate metabolite monoethyl glycinamide increased toxicity, xylene, followed by amide hydrolysis, excreted in the urine, about 10% in the base amount of discharge, a few appeared in bile.
Cardiac output decreased.
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