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ISO9001 Approved Local Anaesthetic Drug Levobupivacaine HCl For Killing Pain

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ISO9001 Approved Local Anaesthetic Drug Levobupivacaine HCl For Killing Pain

China ISO9001 Approved Local Anaesthetic Drug Levobupivacaine HCl For Killing Pain supplier
ISO9001 Approved Local Anaesthetic Drug Levobupivacaine HCl For Killing Pain supplier ISO9001 Approved Local Anaesthetic Drug Levobupivacaine HCl For Killing Pain supplier

Large Image :  ISO9001 Approved Local Anaesthetic Drug Levobupivacaine HCl For Killing Pain

Product Details:

Place of Origin: WUHAN
Brand Name: Yuancheng
Certification: ISO9001
Model Number: Pegylated Mechano Growth Factor,PEG MGF

Payment & Shipping Terms:

Minimum Order Quantity: 10 gram
Price: Negotiable
Packaging Details: Discreet packaging
Delivery Time: About 4-7 working days
Payment Terms: T/T, Western Union, MoneyGram,bitcoin
Supply Ability: 300KG/Month
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Detailed Product Description
Product Name: Levobupivacaine HCl CAS NO.: 27262-48-2
Molecular Formula: C18H28N2O.HCl Molecular Weight: 324.89
Assay: 99% Grade: Pharmaceutical Grade
Storage: Closed, Confined And Shading Preservation Usage: Local Anesthetic

Local Anaesthetic Drug Levobupivacaine HCl for killing Pain

 

Product Name Levobupivacaine hydrochloride
CAS 27262-48-2
MF C18H28N2O.HCl
MW 324.89
Assay 99%
Appearance white powder
Grade Pharmaceutical Grade

 

ISO9001 Approved Local Anaesthetic Drug Levobupivacaine HCl For Killing Pain

 

Description:

 

Levobupivacaine is a local anaesthetic drug belonging to the amino amide group. It is the S-enantiomer of bupivacaine,it is a reversible neuronal sodium channel inhibitor, used as a long-acting local anesthetic.

 

Levobupivacaine hydrochloride is commonly marketed by Abbott under the trade name Chirocaine.

 

Compared to bupivacaine, levobupivacaine is associated with less vasodilation and has a longer duration of action. It is approximately 13 percent less potent (by molarity) than racemic bupivacaine and has a longer motor block onset time.

 

Levobupivacaine is an amide-type local anaesthetic. Levobupivacaine acts via blockade of voltage-sensitive ion channels in neuronal membranes, preventing transmission of nerve impulses. Localised and reversible anaesthesia is produced by interference with the opening of the sodium channel, which inhibits conduction of the action potential in nerves involved in sensory and motor activity and sympathetic activity. Levobupivacaine displaces 3H-BTX from sodium channels of rat brain synaptosomes with IC50 of 2.9 μM and Hill coefficients of 1.2. When cell membrane is held at -80 mV, -70 mV, -60 mV or -100 mV, Levobupivacaine shows tonic inhibition of sodium channel in GH3 cells with IC50s of 132.1, 37.6, 21.6 and 264 μM, respectively. Levobupivacaine depresses action potential of isolated axon in vitro. Levobupivacaine (1mM) depresses action potential amplitude and maximal rate of rise of action potential (dV/dtmax) in the crayfish giant axons with value of 88 and 81 respectively, after perfusion for 15 min. [3] Levobupivacaine also displays activity on cardiac ion channels. In isolated ventricular myocytes, the apparent affinity for inactivated state of the sodium channel is 4.8 μM for Levobupivacaine, with a calculated KD of 39μM. On inhibition of cardiac delayed rectifier potassium channels (hKv1.5), the steady-state block for Levobupivacaine (20 μM) is 31%, with a calculated KD of 27.3 μM. Levobupivacaine may also inhibit cardiac calcium channels. 10 μM Levobupivacaine produces a 50% decrease in contractile force of guinea-pig papillary muscles.

 

Levobupivacaine has similar nerve blocking potency with bupivacaine. Levobupivacaine at a dose of 0.125%, inhibits motor and nocifensive pinch responses with maximum %MPE of 99 and 68 respectively, and inhibits the duration of deficits of motor and nocifensive pinch responses (60 and 30 , respectively) after sciatic nerve block.

 

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Good quality is one of our secret success, welcome order the samples, MOQ just 10 grams.
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